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Nitrogen metabolism reduces ammonia accumulation and avoids mitochondrial oxidative damage through urea cycle and glutamine synthesis ( + enhances the SIRT1/Nrf2 axis to promote glutathione synthesis, and C O A supports fatty acid metabolism to reduce lipid peroxidation), so as to jointly build a multi-level antioxidant network

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One major obstacle is the difficulty in translating animal model results into human clinical settings due to species-specific variations in drug metabolism, pharmacokinetics, and the complexity of arrhythmias in human patients