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Given the essential role of the mitochondria in senescent cells and in regulating the SASP, dysfunctional mitochondria may serve as an important therapeutic target to mitigate the detrimental effects of cellular senescence and the SASP on vascular function

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In another aspect of the invention, one or more NNMT inhibitors (such as a cation of Formula I or IA, or otherwise disclosed herein) is contacted with a cell expressing NNMT followed by contacting said cell expressing NNMT with one or more chemical entities (e.g., nicotinamide riboside, nicotinamide mononucleotide) that modulate intracellular NAD+ levels to provide muscular therapy